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HEART ATTACK: EMERGENCY MEASURES WITHIN ORTHODOXY
For those faced with emergency heart surgery, are there orthodox treatments which might avoid the risks of invasive treatment modes?
Experts conform that it is not the heart attack which causes heart muscles to die but the starvation of the tissues which ensues in the vital hours and days after it. Harking back to the earlier description of what happens to that part of the heart muscle which has been cut off from its supply of nutrients and oxygen, usually by a spasm or blockage in one of the coronary arteries, it can be seen that distorted chemical reactions occur which attract calcium into the cells which further distorts reactions.
Recent research from the University of Arkansas (and elsewhere) supports the views that many would use an emergency infusion of EDTA at this time, since it is an expert calcium blocker. However there are calcium blockers within traditional medicine which will work too. Dr Morton Walker writes: ‘Any one of the chelating agents, such as ascorbic acid, Ringer’s lactate (containing lactic acid) or other weak organic acids acceptable for intravenous infusion, will help reverse the process of heart-muscle disintegration and protect the cells against dying, clots from forming, and arrythmias from developing.’ He adds, ‘of course, EDTA works most swiftly and effectively, however a cardiologist unfamiliar with its application can employ another safe chelating agent.’
As long ago as 1981 there were orthodox doctors who supported this view. In an address to the Chicago Heart Association in February of that year, Dr Maseri, then Professor of Cardiology at the University of London’s Royal Postgraduate Medical School, opined that three-quarters of all bypass operations, emergency or otherwise, were unnecessary. He believed that most heart attack patients could be stabilized just as well with drugs such as calcium channel blockers.
‘It is true that arteries narrowed by fatty deposits cause some heart attacks,’ Dr Maseri said, ‘but there is also evidence of another cause – spasms of the arteries. These spasms, or convulsive contractions, can occur in either narrowed arteries or in arteries free of fatty deposits, and they can be treated by the [then] new drugs.’
Dr Fritz Schellander, who runs the Liongate Chelation Clinic in Tunbridge Wells, in Britain, offered the illustrations presented here as a visual example of this calcium-induced spasm of a coronary artery. His belief, as it is with all chelation therapists, is that ultimately EDTA could be used more in an emergency capacity to stabilize the metabolic condition underlying the spasm. Its prime role is as a calcium blocker but it also has the facility to remove other metals such as iron and copper which sometimes appear as an additional factor during faulty cell metabolism.
EDTA is also safe. When it is considered that EDTA is the solution into which a heart is put when it is taken from a donor, to be kept there until it is transplanted into a recipient, it can hardly be called otherwise.
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